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That's not so dissimilar to my experience in 2025 with whatever USB drives I can scrounge up around the house!

The issue isn't really Android, it's the touchscreen and the way the UX is a regression from many analog single-purpose devices.

If you gonna have a single-purpose device - make it analog (or close to analog)!

Don't give it a perceptible boot-time and all the other flaws that come with general-purpose computing. Don't make the user have to "wake up the device", let alone have to visually confirm that it is woken-up, before they can switch to the next song.


I loved the buttons on the Galaxy Spica https://www.gsmarena.com/samsung_i5700_galaxy_spica-pictures...

Now that I think about it, going no-buttons might have been a driver towards larger screens. Having at least a few buttons seemed to make it much less necessary.


There is plenty more for a cardiologist to do even if they solve the most pressing current problems. Hard to run out of puzzles in biology.


> High LDL is correlated with the development of heart disease, but it does not cause heart disease.

You realize this sentence is an oxymoron?

Unless you meant to say "it does not cause the development of heart disease". I agree correlation is not causation.


I don't think it is. Something can either be correlated and causal or correlated and non-causal. It makes sense to talk about which.


> You realize this sentence is an oxymoron?

No it isn't.

Think of heart disease as slow, long-term damage to the cardiovascular system, and cholesterol is what the body uses as a bandaid.

If you have a lot of LDL cholesterol available, your body will use a lot of it, and you'll have stiffer arteries. If you don't have much available, it takes longer for the bandaids to build up.

This is one of the reasons statins reduce the number of heart attacks, but don't always seem to reduce all-cause mortality.


The band aid analogy doesn’t make sense when we consider the MR studies showing the lower your genetically determined LDL-c, the lower your risk of CVD. If everything was randomised except the number of band aids, why would having fewer band aids result in lower CVD risk?

> This is one of the reasons statins reduce the number of heart attacks, but don't always seem to reduce all-cause mortality.

That’s one potential explanation, but I don’t think it’s the most likely one. We tend to see non significant ACM in smaller, less powered trials, or those with lower LDL-c lowering. ACM is simply a less sensitive endpoint - if you have a treatment that reduces CVD incidence, then the “CVD incidence” endpoint will give you significant results with fewer CVD event differences between study arms compared to ACM since your power to detect differences is diluted by other fatal events that aren’t affected by statins (cancer, motor accidents etc).


You realize correlation does not imply causation?

Edit: this was written before OP edited their comment


> tell the AI to write in the style of a HN commenter, or whatever.

This might turn out to be one of those tricks like Ctrl+C Ctrl+V that surprisingly few people discover.


...ChatGPT? Such an odd take, to point at weather being variable.

This is a coastal city at a fairly run-of-the-mill latitude, people build functional bike networks in much worse.


I'll point you to my prior comment re:bike-commuting in D.C. versus the same in Boulder, Colorado. https://news.ycombinator.com/item?id=46367940

There needs to be an entire wholesale change in both infrastructure and culture to make bike-commuting workable in most extant cities.

Relatively speaking, the infrastructure is the easy part.

I think we'll get to the heat death of the universe before bike-commuting in Houston, Texas would ever be "a thing".


A discussion is held between people who don't (initially) believe each other. Nothing odd about it.


Not clear in your example if you have a to-dont list, or just decide "don't" in the moment.


You mean... an aha list?


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